A pulse dose system is the most common used portable oxygen concentrator because of its light weight and long battery life. While the difficulty of battery capacity is less than with the dose system, you should consider the battery as a primary issue. The weight of the handheld Oxygen Concentrator is only 4.4 oz and is easy to handle. On average, a 1,000mL-style portable oxygen concentrator takes 3 to 4 hours to recharge. Once you reach your capitalization time, you can hold it on your finger tip with the strap still on and use it as you would a complete 5-stage Oxygen Dosing Device.
Using the Portable Oxygen Concentrator
The Oxygen Concentrator contains a 5-stage oxygen buffer system which consists of a primary oxygen buffer, an excess dissolved oxygen tank and two supplemental oxygen tanks. In its on-board reflux port, it is capable of reducing oxygen emissions to 1.6%, which is higher than most on-board applications such as plants or rust-proofing.
With an available capacity of 125 L, the Oxygen Concentrator provides ample oxygen for a variety of vehicle applications.
The Compressor for the Oxygen Concentrator is a volatile gas with an approximate molecular mass of 9.6 grams per mole. By extracting oxygen from fuel and air to increase the percentage of dissolved oxygen in the air, water and the water used to clean the air as part of the therapy process, oxygen content in the air is lowered. Unlike the pressurized systems used for breathing and for filtering oxygen from food and drink, oxygen has to be exposed to unshielded oxygen within a respirator to be effective.
Much less is required for the treatment of patients with chronic illness, in which the oxygen content in the air is very low. Oxygen concentration in the air varies widely across the globe. In the Northern Hemisphere, relatively high oxygen concentrations in cold winter months mean that the influx of incoming oxygen from the outside is slower than it is in warmer months. During winter months, rapidly flowing high-oxygen air tends to pull more oxygen out of the lung than does rapidly flowing low-oxygen air. Thus, colder air holds less oxygen;, low-oxygen air holds more oxygen;, if breathed in excessively, the o2 concentration can be significantly less than in warmer air;, if breathed in, the lungs often are deprived of their supply of o2, and the patient begins to exhibit symptoms of hypoxia.
In some cases, possibly similar to the symptoms of metabolic acidosis, if oxygen continues to be retained in the lungs, one end of the gas tube is damaged and carbon dioxide can collect within the system. Even if a complete hypercapnic air is able to be breathed, the patient may experience carbon dioxide retention, and will likely develop cough and dyspnea.
Up to 30% of sick military personnel with high-altitude illness receive oxygen treatment in the form of a low-oxygen gas-air mixture, some of which is meant to be inhaled (14, 15).
In high-altitude patients, early understanding of underlying disease processes can lead to effective treatment, and ultimately improvement (8, 8, 8, 18, 16, 18, 20, 24). However, as a point of reference, one study found that air forced into one patient’s lungs for 10 minutes had the same effect as 1,000 doses of oxygen (25). Further, a high-altitude patient in a community setting who receives a mixture of oxygen and an anesthetic gas is still considered to have 100% oxygen saturation, despite normal levels of oxygen concentration in the surrounding air (20, 26). In these circumstances, hypoxia can impact overall health and can jeopardize life if treatment is not delayed.
Ozone is often used in prehospital oxygen delivery to help maintain oxygen saturation to ensure that the patient has adequate oxygen saturation. Therefore, precautions should be taken to protect other vital organ systems, especially those systems dependent on ATP generation, including the brain, lungs, and other capillaries in addition to the respiratory center.
Dementia-Risk Assessment: Screening for Mild Cognitive Impairment.
Persons with mild cognitive impairment should be screened for dementia. Individuals with mild cognitive impairment are considered at increased risk for developing Alzheimer’s disease and Alzheimer’s disease-related dementia. These individuals also have higher rates of dementia compared with their normal-risk counterparts.
Further, cognitive impairment can lead to slower response times in the operation of vehicles and machinery.
In addition, individuals with Alzheimer’s disease may experience such conditions as irritability, mood swings, depression, confusion, loss of speech, difficulty eating and sleeping, and loss of coordination and balance.
In some areas, the rate of cognitive impairment in individuals with mild cognitive impairment is significantly higher than in adults without this degree of impairments. Thus, it is possible that the occurrence of light intoxication may not be predictable in the general population of older people with moderate-to-severe Alzheimer’s disease.
With respect to prescription medicines, it is not known whether for-going light is the safest approach to managing dementia and cognitive impairment associated with Alzheimer’s disease. A 2014 study reported the beneficial effects of light therapy for mild cognitive impairment (MCI) on psychological well-being in healthy elderly people.
However, in the present study, no significant effects of light therapy were observed for cognitive functioning, emotionality, health behavior, quality of life, or the ICU discharge rate in this population. Because no safety data on ICU ventilator use for ICU resuscitation and for Alzheimer’s disease have been published,11 we consider light therapy to be safe for the treatment of these conditions.
Assessment of Patient Outcomes in The Epilepsy and Drug Abuse Program
As previously mentioned, the placebo-controlled trial design was not appropriate for evaluating the potential effects of EMT in patients with epilepsy. Nevertheless, in this primary outcome assessment (ie, the responses to a medical evaluation) we used an intention-to-treat approach in which half of the patients (N=28) received EMT alone and the other half received placebo (N=27).
The primary outcome was seizure reduction at 24 months (symptoms resolved), and other secondary outcomes (ie, measures of quality of life) were all assessed at a 2-year follow-up.
Although EMT led to significantly better quality-of-life scores at both 12-month and 24-month follow-up, there was no difference in seizure reduction at 24 months for the 2 groups. The relatively small number of patients who took EMT contributed to the results.
Oxygen Concentrators – Summaries for Consumers
This potential risk includes the potential risk of oxygen deprivation. Some airways constrict due to anesthesia, exposing a patient to significant levels of oxygen. For that reason, oxygen concentrators should be placed only in the right lung and sealed completely and securely.
Symptoms and Signs
The presence of an oxygen concentrator in a lung can lead to a variety of symptoms and signs. Signs that can result include the following:
Shortness of breath
Shortness of breath can often be caused by both pneumonia and thoracic duct obstruction. In some cases, shortness of breath is a result of infection. In some cases, shortness of breath is a result of infection.
Airway obstruction may occur even if the oxygen concentrator does not cause symptoms. The airway may be blocked by tiny particles of dust, dirt, and pollen that irritate the nose, throat, and upper airway. Airway obstruction can be severe and cause difficulty breathing. If the airway is blocked by the oxygen concentrator, you may gag or vomit. If the airway is blocked by the oxygen concentrator, you may gag or vomit.
If you do not respond, you may assume that the oxygen concentrator is not present.